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Uterine polyps 2

UTERINE POLYPS AND CANCER: Natural treatment

By | On 26/12/2019 | Comments (0)

UTERINE POLYPS AND CANCER

A  polyp can sometimes seem like a fibroid uterus  ( polyp fibrous) or cancer endometrial ( polyp atypical, irregular). Conversely, cancer endometrial can sometimes look like a  polyp. The combination of a  polyp to cancer endometrial is more common after menopause. However, Dawasanté experts provide you with a natural treatment to cure uterine polyp cancer. Click on the image below to discover this natural treatment.

Uterine Polyps and Cancer: Natural Treatment

To contact our experts please call or write to us on the following number, tel / WhatsApp:  https://wa.me/22996374527

 The uterine polyps are growths that develop at the expense of the lining of the uterus (endometrium), or at the expense of the lining of the cervix (endocervical).

These growths are benign tumors. They can be single or multiple. A polyp can be "pedicle" (comprising an insertion foot) or sessile (broad implantation base). It can be "fibrous" or "mucous". By definition, the development of a  polyp occurs at the expense of a mucous membrane. This is why, in addition to the uterus, certain organs or areas of the human body can be invaded by this type of tumor such as the bladder,  colon,  stomach,  vagina, etc.

The uterine polyps are outgrowths attached to the inner wall of the uterus which extends into the uterine cavity. Cellular proliferation in the lining of the womb (endometrium) results in the formation of uterine polyps, also called endometrial polyps. These polyps are usually non-cancerous (benign), although some can be cancerous or turn into cancer (precancerous polyps).

A  uterine polyp can look like a  fibroid or a cancerous tumor, so be careful not to get the wrong diagnosis. Cancerization of the polyp is possible.

All examinations performed should show whether the uterine polyps are benign or not. Thus, after removing these growths, a complete analysis of them is required. Postmenopausal women are prone to endometrial cancer combined with uterine polyps.

Two forms of treatment are possible to treat polyps, the choice is based on their benignity, size, appearance, etc. Those that are small and less worrying may go away as a result of hormonal drug therapy. With this treatment, the patient takes progestins. In most cases,   operative hysteroscopy is necessary, it consists of the removal of the polyps. This form of treatment should be carried out with great care, especially in women who wish to have children later. For postmenopausal women, the risk of recurrence being high,  endarterectomy is recommended.

Endometrial cancer is a cancer of the inside of the uterus, where the endometrium is the lining that lines the inside of the uterus. In women with cancer at this level, endometrial cells multiply abnormally. Endometrial cancer usually occurs after menopause, but 10 to 15% of cases affect premenopausal women, including 2 to 5% of women under 40 years old.

A significant proportion of endometrial cancer is thought to be attributable to an excess of estrogenic hormones produced by the ovaries or supplied by the outside. The ovaries produce 2 types of hormones during the female cycle:  estrogen and progesterone. These hormones act on the endometrium throughout the cycle, stimulating its growth and then its expulsion during menstruation. An excess of estrogen hormones would create an imbalance conducive to the poorly controlled growth of endometrial cells.

Several factors can increase estrogen levels, such as obesity or hormone therapy with estrogen alone. This type of hormone therapy is therefore reserved for women who have had the uterus removed or hysterectomy who are no longer at risk of endometrial cancer. For more information, see the People at risk and Risk factors sections.

For some women, however, endometrial cancer does not appear to be caused by a higher level of estrogen.

Other causes are involved in endometrial cancer, such as advanced age, overweight or obesity, genetics, hypertension, etc.

Sometimes cancer occurs without a risk factor being identified.

Like any mucous membrane, the endometrium is made up of surface cells called “epithelial” and supporting tissue, the “chorion”. More than 90% of endometrial tumors develop at the expense of the epithelium. This category includes cancerous lesions such as adenocarcinomas and carcinosarcomas, pre-cancerous lesions such as atypical hyperplasias, endometrial polyps, and lesions induced by tamoxifen. Adenocarcinomas, the most common form, are developed from "glandular" type epithelial cells. 

Other metabolic factors: diabetes and glycemic load

Diabetes is a risk factor for endometrial cancer. The risk of developing this cancer is twice as high (significant relative risk) in diabetic patients than in people without diabetes (Larsson, 2006).

It is the development of insulin resistance that is responsible for the increased risk of cancer. Insulin is then in large quantities in the blood and will be able to stimulate growth factors acting on the endometrium. The precise mechanisms are not well known. This is confirmed in the 2013 WCRF / AIRC report. This same international report also found that increased glycemic load (which estimates the ability of foods eaten to increase blood glucose) is a risk factor for endometrial cancer with a probable level of scientific evidence to account. based on current scientific data; these are not as convincing as for obesity.

Early puberty and late menopause

Age at puberty determines when the ovaries start producing estrogen, and menopause determines when they stop producing estrogen. Earlier puberty or late menopause, therefore, exposes a woman to estrogen for a longer period of her life and therefore represents a risk factor.

Not having had a child (nulliparity)

The menstrual cycle of women experiences a period during which the functional area of ​​the endometrium collapses. In order to accommodate the future fertilized egg, the endometrium must regenerate. For this, estrogen plays a role in stimulating cell growth in the endometrium.

During pregnancy, there is no ovulation for 9 months. The estrogen stimulation does not stop, however, but is much less than if there had been 9 ovarian cycles. Thus, women without children are exposed to greater amounts of estrogen: this is considered a risk factor for the development of endometrial cancer (Lochen, 1997).

This is sort of how polyp can be the root cause of cancer or various tumors.

To contact our experts please call or write to us on the following number, tel / WhatsApp:  https://wa.me/22996374527

 

 

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